◆个人简历
王静，博士，教授，博士生导师（硕士、博士招生单位均在国家重点实验室），四川省学术与技术带头人。获得了国家优秀青年科学基金、四川省杰出青年科学基金、霍英东教育基金、天府青城计划等多个人才项目的资助，主研完成教育部自然科学研究成果奖一等奖（第2，2025）、四川省自然科学奖一等奖（第3，2021），荣获教育部霍英东青年科学奖一等奖、中国作物学会青年科学奖、四川省杰出青年科学技术创新奖、四川省青年科技奖、中国作物学会青年科技奖等科技奖励。主要从事水稻高产抗病分子机制的研究，在Science、Nature Plants、Cell Research等学术期刊发表SCI论文40余篇，多篇论文入选ESI热点论文和高被引论文、被F1000推荐为新发现，研究成果分别被遴选为2017年中国农业重大科学进展、2018年中国农业科学进展。
◆发表论文
1.A single transcription factor promotes both yield and immunity in rice. Science, 2018, 第一作者兼通讯作者.
2.Pathogen-induced H2O2 triggers phosphorylation of the RNA-binding protein RRM2R to suppress intron intention of OsMAPKKK18 for orchestrating immunity and growth in rice. Molecular Plant,2026, 通讯作者.
3. Rice transcription factor bHLH25 confers resistance to multiple diseases by sensing H2O2.Cell Research,2025, 通讯作者.
4. OsHYPK/NatA-mediated N-terminal acetylation regulates the homeostasis of NLR immune protein to fine-tune rice immune responses and growth. Cell reports, 2025,,通讯作者.
5. U-box E3 ubiquitin ligase OsPUB41 represses rice blast resistance by promoting degradation of OsPALs, New Phytologist,2025, 通讯作者.
6. Dynamics of Epitranscriptomes uncover translational Reprogramming directed byac4C in rice during pathogen infection. Nature Plants,2024,通讯作者.
7.Fine-tuning of IPA1 transactivation activity by E3 ligase IPI7-mediated non-proteolytic K29-ubiquitination during Magnaporthe oryzae infection. Nature Communications,2024,通讯作者.
8. E3 ubiquitin ligase IPI1 controls rice immunity and flowering via both E3 ligase-dependentand and -independent pathways. Developmental Cell,2024,通讯作者.
9. D53 represses rice blast resistance by directly targeting phenylalanine ammonialyases. Journal of Integrative Plant Biology,2024,通讯作者.
10.Tissue-Specific Ubiquitination by IPA1 INTERACTING PROTEIN1 Modulates IPA1 Protein Levels to Regulate Plant Architecture in Rice. Plant Cell, 2017, 第一作者.